Role of rad23 and dsk2 in nucleotide excision repair and spindle pole body duplication article pdf available march 2007 with 56 reads cite this publication. Surprisingly ddi1 and dsk2 do not show any redundancy but the triple deletion shows an synthetic defect suggesting that rad23 has at least two different roles in cell cycle progression during g2 m in addition we found that these putative roles do not include a role in spb duplication which contradicts a previously reported study 3. One class is the ubl uba family of proteins fig 1 with the rad23 dsk2 and ddi1 proteins and homologues being most extensively studied this family of proteins is involved in a variety of additional cell processes such as nucleotide excision repair ner spindle pole body duplication and cell growth. Rad23 proteins have an important role in nucleotide excision repair ner 12 yeast rad23 forms a high affinity interaction with rad4 and the dimer can preferentially bind damaged dna 3 4
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